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Anterior Cruciate Ligament Injury and Your PetInjury to the Anterior Cruciate Ligament (ACL) or Cranial Cruciate Ligament (CCL) is one of the most common causes of rear leg lameness in dogs. The condition is uncommon in cats. The function of the ACL is to constrain the knee joint by limiting internal rotation, forward displacement of the tibia relative to the femur, and to prevent hyperextension. Rupture of the ACL can be either complete or partial. Untreated patients with ACL rupture will develop degenerative joint disease (DJD) or arthritis in the affected knee within a few weeks and severe changes within a few months. In over 50% of ACL injuries there is also damage to the medial meniscus. The medial meniscus is a cartilage cushion between the femur and the tibia. CAUSES Injury to the ACL can either be degenerative (chronic) or acute (traumatic). Causes of ligament degeneration include aging, conformational abnormalities (bowlegged, knock-knee, straight knee or hock, patellar luxation, caudal sloping of the tibial plateau), disuse related to sedentary life style or limb immobilization, and immune mediated (arthritis, synovitis). Larger dogs (over 30 pounds) are more likely to suffer from ligament degeneration related to aging than are small dogs. DIAGNOSIS Diagnosis of
either a partial or complete ACL rupture is usually made based on the history
and physical examination. Clinical signs are related to the degree of rupture
(partial vs. complete), the mode of rupture (acute vs. chronic), the presence
of meniscal injury, and the severity of inflammation and DJD. Athletic or
traumatic (e.g. hit-by-car, stepped-in-hole) events usually precede acute ACL
injuries that result in non-weight bearing lameness (partial-toe-touching or
complete). Normal activity that results in acute lameness is suspicious of
degenerative ACL rupture. Mild to marked intermittent lameness that has been
going on for weeks to months is consistent with partial ACL tears progressing
to complete rupture. Presence of a cranial drawer sign (cranial movement of
the tibia while the femur is held motionless) is diagnostic of ACL rupture.
The knee joint may be swollen due to fluid accumulation or there may be a
noticeable thickening of the joint capsule on the medial side (medial
“buttress”). Hindlimb muscle atrophy may be present in chronic cases. TREATMENT
Dogs less than 30 pounds can be treated conservatively with 85% being improved
or normal by 6 months. Only 20% of dogs greater than 30 pounds are improved
by 6 months when treated conservatively. Therefore, it is recommended that
medium-to large-breed dogs should have surgery. Surgery is recommended for
all dogs to speed recovery, to prevent degenerative changes, and to
enhance/restore function. PROGNOSIS Regardless of surgical technique, the success rate is approximately 85%. Second surgeries are required in 10 to 15% of patients due to meniscal damage or implant failure. Regardless of the method of treatment, DJD is common. Return to complete athletic function is uncommon. 20 to 40% of dogs with unilateral ACL rupture will rupture the ACL in the other knee within 17 months.
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